Path ID: DB00555_MESH_D012640_1

Concepts
Identifier | Name | Type |
---|---|---|
MESH:C047781 | lamotrigine | Drug |
InterPro:IPR001696 | Voltage gated sodium channel, alpha subunit | GeneFamily |
GO:0061530 | aspartate secretion, neurotransmission | BiologicalProcess |
GO:0061535 | glutamate secretion, neurotransmission | BiologicalProcess |
HP:0020219 | Motor seizure | PhenotypicFeature |
MESH:D012640 | Seizures | Disease |
Relationships
NOTE: predicates are annotated in Biolink Model (v1.3.0)
Subject | Predicate | Object |
---|---|---|
Lamotrigine | NEGATIVELY REGULATES | Voltage Gated Sodium Channel, Alpha Subunit |
Voltage Gated Sodium Channel, Alpha Subunit | POSITIVELY REGULATES | Aspartate Secretion, Neurotransmission |
Voltage Gated Sodium Channel, Alpha Subunit | POSITIVELY REGULATES | Glutamate Secretion, Neurotransmission |
Aspartate Secretion, Neurotransmission | POSITIVELY CORRELATED WITH | Motor Seizure |
Glutamate Secretion, Neurotransmission | POSITIVELY CORRELATED WITH | Motor Seizure |
Motor Seizure | POSITIVELY CORRELATED WITH | Seizures |
Comment: This drug may also affect GABA-mediated synaptic transmission (https://go.drugbank.com/drugs/DB00555#BE0000013). However this is rather contentious, so it has not been annotated as such in here. Besides if the drug does modulate gamma-aminobutyric acid receptor it’s a rather weak inhibition seen in animal models (yet to be determined in humans). Note that the drug could be contraindicated for treatment of seizures as it can exacerbate them (https://pubmed.ncbi.nlm.nih.gov/9596203/). Finally, note that this disease is denoted as Tonic-clonic seizure in the original file but known as Seizures in MESH.
Reference:
- https://go.drugbank.com/drugs/DB00555#mechanism-of-action
- https://en.wikipedia.org/wiki/Lamotrigine#Mechanism_of_action
- [https://en.wikipedia.org/wiki/Glutamate_(neurotransmitter)#Disease,disabilities,_and_pharmacology](https://en.wikipedia.org/wiki/Glutamate(neurotransmitter)#Disease,_disabilities,_and_pharmacology)